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We followed 42 patients with clinically defined pure sensory neuropathy of acute or subacute onset for 2 to 35 years.The symptoms began in the upper limbs in 23 patients,in the lower limbs in 13,symmetrically in all 4 limbs in 4,and the face was 1st affected in 2.For 19 patients,the symptoms began asymmetrically.Electrophysiologic testing typically showed absence of sensory potentials.Spinal fluid was usually acellular with a normal protein level.Sural nerve biopsy in 22 patients showed loss of large myelinated fibers and axonal atrophy without inflammation.Six of the patients died:4 of unrelated caused and 2 of subdural hemorrhages.Only 2 patients had severe functional impairment.Twenty-two had significant sensory deficit but were able to carry out most of their usual activities. In 8,the symptoms had resolved completely.The acute,often focal onset suggests an immune-mediated or vascular process at the level of the posterior root or dorsal root ganglion. |
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areflexia
face,numbness of
ganglionitis
girdle sensation
mortality
nerve biopsy
nerve conduction studies
neuronopathy,sensory
numbness,generalized
polyneuritis,acute sensory
prognosis
sensory polyneuropathy
wheelchair
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